“Iron deficiency can impair red blood cell production and tissue respiration by reducing the amount of proteins that carry and consume oxygen (hemoglobin and myoglobin).
When is Ascofer used?
iron deficiency conditions associated with blood loss (after injuries, chronic bleeding, surgery, dialysis, in women with heavy menstrual bleeding);
states of increased body need for iron (in children during periods of intensive growth, during pregnancy and breastfeeding);
conditions associated with malabsorption (after gastrectomy, malabsorption syndrome, persistent diarrhea) or with a diet containing too little iron.
Active ingredient: Ferrozi gluconas.
Compound
Active substance The active substance of the drug is iron(II) gluconate in a dose of 200 mg, which corresponds to 23.2 mg of iron ions.
1 film-coated tablet contains 200 mg of iron(II) gluconate.
other ingredients Other ingredients (excipients): ascorbic acid, talc, potato starch,
stearic acid, propylene glycol and AquaPolish P red coating consisting of: hypromellose,
hydroxypropylcellulose, macrogol, triglycerides of medium-chain saturated fatty acids
, talc, titanium dioxide (E 171), Allura red AC, aluminum varnish (E 129), black iron oxide (E 172),
red iron oxide (E 172).
Ascofer dosage
Medically:
Adults: usually 2 tablets three times a day during or after meals; with a significant deficiency – 2 tablets 4 times a day.
Children over 3 years of age: therapeutically, 4–6 mg of iron per kg of body weight daily in several divided doses.
Preventatively:
Adults: Approximately 60 mg iron (3 tablets) per day in divided doses.
Children: 2 mg per kg body weight per day in divided doses.
To obtain the full therapeutic effect, regular and long-term use is necessary. The duration of treatment should be determined by the doctor.
Directions for use: oral.
When should you not use Ascofer?
Hypersensitivity to the active substance or to any of the excipients.
Ferrous gluconate should not be used for:
blood diseases: hemosiderosis, hemochromatosis (pigmented cirrhosis of the liver, characterized by increased absorption and excessive deposition of iron in the internal organs in the form of hemosiderin);
hemolytic and aplastic anemia;
multiple blood transfusions;
parenteral administration of iron supplements;
porphyria cutanea tarda;
impaired absorption from the gastrointestinal tract;
proliferative diseases;
cirrhosis of the liver.”
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